Abnormal blood cell shapes and children born with polycythemia (a disease in which the proportion of blood volume made up of RBCs increases) can create an overload of RBCs to be processed. Source: Internet
Abnormal adhesion and decreased deformability of RBCs are the main causes of blood vessel occlusion (vaso-occlusion) in SCD. Source: Internet
All major factors regarding RBCs demonstrated some level of damage present for all levels of toxin administered and across all Cry proteins, although there were some clear variances present between different proteins and levels for certain factors. Source: Internet
In individuals with abnormal RBCs, whether due to abnormal hemoglobin molecules (such as Hemoglobin S in Sickle Cell Anemia) or RBC membrane defects – or other problems, the RBC half-life is frequently shortened. Source: Internet
This Hb A 1c level is only useful in individuals who have red blood cells (RBCs) with normal survivals (i. Source: Internet
Antibodies can be highly active and can attack RBCs and bind components of the complement system to cause massive hemolysis of the transfused blood. Source: Internet